Team:Queens

From 2009.igem.org

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<p style="font-size:175%;font-family:corbel;color:#172C4E;font-weight:bold"><i>Project Background</i></p>
<p style="font-size:175%;font-family:corbel;color:#172C4E;font-weight:bold"><i>Project Background</i></p>
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This year, the Queen’s iGEM team is exploring a new synthetic biology approach to treat atherosclerosis. The purpose of our project is to engineer E.coli cells to target and deliver drugs to the site of atherosclerotic plaque. This will be accomplished by designing a binding system that allows E.coli to adhere to plaque, an inducible effector system that produces and releases drugs to site of plaques, and a termination system that detaches the E.coli from the plaque and triggers self-destruction. For a more detailed explanation and breakdown please see our Project page.
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This year, the Queen’s iGEM team is exploring a new synthetic biology approach to treat atherosclerosis. The purpose of our project is to engineer <i>E.coli</i> cells to target and deliver drugs to the site of atherosclerotic plaques. This will be accomplished by designing a binding system that allows <i>E.coli</i> to adhere to plaque, an inducible effector system that produces and releases specific drugs to site of plaques, and a termination system that detaches the <i>E.coli</i> from the plaque and triggers self-destruction. For a more detailed explanation and breakdown please see our Project page.
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Last Updated: May 26, 2009 by Fr<sub>3</sub>P</p></center>
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Last Updated: July 31, 2009 by Fr<sub>3</sub>P</p></center>
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Revision as of 04:32, 1 August 2009



Team Background

More information yet to come!


Project Background

This year, the Queen’s iGEM team is exploring a new synthetic biology approach to treat atherosclerosis. The purpose of our project is to engineer E.coli cells to target and deliver drugs to the site of atherosclerotic plaques. This will be accomplished by designing a binding system that allows E.coli to adhere to plaque, an inducible effector system that produces and releases specific drugs to site of plaques, and a termination system that detaches the E.coli from the plaque and triggers self-destruction. For a more detailed explanation and breakdown please see our Project page.



Last Updated: July 31, 2009 by Fr3P